![]() Conclusion: This study strongly suggests that experiential symptoms are largely dependent upon functional modification of the physiology of the rhinal cortices.įrom the 1850s, Saint-Saëns regularly employed cyclic form: the practice of establishing large-scale relationships (especially in symphonies, chamber works, etc.) by reintroducing materials from earlier movements in later ones. In particular, déjà vu was associated with stimulation of the EC and reminiscence of memories with PC stimulation. Such déjà vu or déjà vécu type responses were produced proportionately more often by stimulation of the EC than by stimulation of the amygdala and hippocampus. Entorhinal and perirhinal stimulations induced classic mesial temporal lobe responses (emotional, dysautonomic) but also more specific responses, particularly the déjà vu phenomenon and reminiscence of scenes. ![]() Results: A total of 280 stimulations were analyzed. All patients had electrodes that sampled the rhinal cortices, amygdala, and hippocampus. ![]() Stimulations were performed in a group of 24 patients with epilepsy, during stereo-electroencephalographic (SEEG) recordings in the setting of presurgical evaluation. Methods: The authors studied the symptoms evoked by direct electrical stimulations of PC and EC in comparison with those obtained after stimulation of the amygdala and hippocampus. The role of sub-hippocampal structures (EC and PC) in inducing these phenomena has not previously been addressed. These phenomena have been attributed to functional alteration of memory networks supported by the medial temporal lobes, principally involving the amygdala and hippocampus. Objective: To study the role of perirhinal (PC) and entorhinal cortices (EC) in dreamy state symptoms (déjà vu and reminiscence of scenes). During the two-year follow-up, the déjà vécu sensations continued unaltered, but all involved were less bothered by them, with the patient’s functioning improving both personally and socially. Our patient and her family were offered psychoeducation, which led to acceptance and improved coping. Proposed to be a functional disturbance of the limbic system with involvement of a network that comprises at least the hippocampus and entorhinal cortex, the pathophysiology of déjà vécu is in need of further elucidation. An extensive psychiatric and somatic work-up ruled out cerebrovascular disease, epilepsy, dementia, psychosis, or intoxication as a potential underlying cause. We here describe an 84-year-old woman with idiopathic, partial déjà vécu, where her symptoms were limited to people and events. ![]() Having the delusional conviction that this sensation is true, patients frequently exhibit recollective confabulation. Given that this is a common feature of the déjà vu experience, one potential way for an experiencer to psychologically resolve the bizarreness of this (and the perhaps ensuing sense of losing touch with reality) is to attribute the sense of prediction to a paranormal cause, such as believing that one is being assisted by spirits or exhibiting psychic ability.ĭéjà vécu is an extremely rare type of identifying paramnesia characterised by the ongoing sensation of having experienced things before. As discussed in Chapter 12, reports of the subjective experience of déjà vu are commonly accompanied by a sense of knowing what will happen next (Burnham, 1889 Chari, 1964 Dugas, 1894 Holmes, 1891 Jensen, 1868, cited in Marková & Berrios, 2000Kraepelin, 1887 McKellar, 1957 Osborn, 1884 Reed, 1979 Sanders, 1874, cited in Marková & Berrios, 2000 Sno & Linszen, 1990 Titchener, 1924 Ward, 1918), and some even incorporate this dimension into their definition of déjà vu (Carrington, 1931 Dugas, 1894 Jensen, 1868 Krijgers Janzen, 1958 Lalande, 1893 Myers, 1895 Ward, 1918 Sno, 2000 Titchener, 1924) (see Table 1.2). ![]() 255-256) The relationship between déjà vu and so-called precognition also has a long history. ![]()
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